Abstract: Mitochondrial function is fundamental to neuroregeneration, particularly in neurons, where high energy demands are essential for repair and recovery (Patrón and Zinsmaier, 2016; Beckervordersandforth et al., 2017; Iwata et al., 2023). Mitochondrial dysfunction, characterized by an imbalance in ATP levels and excessive production of mitochondrial reactive oxygen species, is a key factor that impedes neural regeneration in neurodegenerative diseases and after neuronal injury (Han et al., 2016, 2020; Zheng et al., 2016; Zong et al., 2024). Targeting mitochondrial health is therefore crucial for advancing therapeutic strategies in neuroregeneration.