Abstract: Cochlear implants (CI) are widely used to provide auditory rehabilitation to individuals with moderate to severe sensorineural hearing loss (Eshraghi et al., 2012). The scala tympani (ST) of the cochlea is the site of implantation of the intracochlear electrode array. In a healthy, normal ear, the cell bodies of the spiral ganglion neurons (SGNs) reside in Rosenthal’s canal, a small cavity adjacent to the ST. SGNs have a peripheral neurite that projects to the hair cells on the basilar membrane of the organ of corti, and a central axon that projects to the brainstem via the auditory nerve (Landry et al., 2013). From SGN cell bodies, the dendrites extend through the modiolus and the osseous spiral lamina to make synaptic contact with hair cells in the organ of corti (Rusznák et al., 2009). In severe to profound deafness, the cochlea has few to no hair cells (Shibata et al., 2010). A CI helps overcome the problem of functional hair cells by directly stimulating the SGNs in the inner ear via short biphasic electric pulses (Li et al., 2017).