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    01 December 2018, Volume 13 Issue 0 Previous Issue    Next Issue
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    Components of Houshiheisan Promote the Recovery of Axon via Regulating Nogo-A/RhoA/Rock2 and Netrin-1/ Rac1/Cdc42 on Ischemic Cerebral Injury in Rats
    Yue Lu, Flora Hsiang, Jiahui Chang, Xiaoquan Yao, Hui Zhao, Haiyan Zou, Lei Wang, Qiuxia Zhang
    2018, 13 (0):  1. 
    Abstract ( 222 )   PDF (2301KB) ( 422 )   Save

    Houshiheisan (HSHS) which is created by Zhang Zhongjing, contains two main components of drugs——wind-dispelling drug (WDD) and deficiency-nourishing drug (DND). The compatibility of WDD and DND has adequately showed the theory of “reinforcing the weak body and expelling the pathogenic factor of excess type”. In this study, we investigated the mechanisms of HSHS and its components in axonal remodeling after cerebral ischemia by observing pathological changes and detecting the expression of some relevant proteins. Rats were randomly divided into 5 experimental groups as follow: sham operation group (Sham, n = 12), pMCAO group (pMCAO, n=15), HSHS group (HSHS, n=15), WDD group (WDD,n=15) and DND group (DND, n=15). All drugs were given by intragastric administration respectively, and
    the dosage of HSHS, WDD and DND were 10.5g/kg, 7.7g/kg and 2.59g/kg. Suture method was used for permanent occlusion of middle cerebral artery (pMCAO) model to simulate focal cerebral ischemia. Neurological function score and balance beam walking test were performed after pMCAO as neuroethological assessments. HE staining and transmission electron microscopy were used for the assessment of neuropathological changes. Immunofluorescence combined with image analysis technique was used to detect the expression of MAP-2. Western blot was conducted to detect the protein expression including amyloid precursor protein (APP), growth associated protein-43 (GAP-43), neurite outgrowth inhibitor protein A (Nogo-A), Rho family small GTPase A (RhoA), Rho-associated kinase 2 (Rock2), netrin-1, Ras-related C3 botulinum toxin substrate 1 (Rac1) and cell division cycle 42 (Cdc42). These results showed that HSHS,WDD and DND exerted their functions in different aspects via down-regulating the expression of APP,Nogo-A, RhoA, Rock2 and up-regulating the expression of GAP-43, netrin-1, Rac1, Cdc42. In conclusion,HSHS, WDD and DND could improve neurological function, reduce the damage of the axon and promote the recovery of axon by regulating Nogo-A/RhoA/Rock2 and Netrin-1/Rac1/Cdc42 after cerebral ischemia.Importantly, these findings could illustrate the synergic mechanism of WDD and DND, and provide experimental evidence for the application of HSHS on stroke. To draw a conclusion of the study, we confirmthat HSHS has a more potent therapeutic effect on focal cerebral ischemia than WDD or DND alone.

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    Effects of decompression joint Governor Vessel electro-acupuncture on rats with acute upper cervical spinal cord injury
    Wang YanLei, QI YingNa, Wang Wei, YI Ping, Yang Feng, Tang XiangSheng, Tan MingSheng
    2018, 13 (0):  1-6. 
    Abstract ( 165 )   PDF (509KB) ( 308 )   Save

    Early decompression is the major therapeutic strategies of the acute spinal cord injury,Governor Vessel electro-acupuncture can improve symptoms of spinal cord injury by inhibiting cell apoptosis and improving the microenvironment of the injured spinal cord. This study will investigate the Governor Vessel electro-acupuncture’s effect on early and late decompression’s rats with acute upper cervical spinal cord injury.We created the models of acute spinal cord injury in rats with 12-hours and 48-hours compression and gave the rats Governor Vessel electro-acupuncture’s therapy for 14 days, the results showed that, compared with the 12-hours compressed rats, the Governor Vessel electro-acupuncture’s therapeutic on the 48-hours compressed rats had a better performance in motor functional recovery, inhibiting cell apoptosis and improving the microenvironment of the injured spinal cord. in conclusion, for the acute spinal cord injury, the Governor Vessel electro-acupuncture has more effective in rats compressed for 48-hours than that for 12-hours.

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    MGMT is down-regulated in rats with all-trans retinoic acid-induced spina bifida aperta independently of promoter DNA methylation
    Henan Zhang,Yi Guo, Wei Ma, Jia Xue, Weilin Wang, Zhengwei Yuan
    2018, 13 (0):  1-8.  doi: 10.4103/
    Abstract ( 95 )   PDF (917KB) ( 187 )   Save

    The loss of DNA repair genes can cause embryonic teratogenicity and lethality, several DNA repair genes have been reported to be associated with neural tube defects (NTD). O6-methylguanine DNA methyltransferase (MGMT), as one of the DNA repair enzymes has been reported in other congenital malformations,but it is less frequently reported in NTDs. DNA damage was assessed by detecting γ-H2A.X in ATRA-induced SBA rats. Real-time PCR (RT-PCR) was used to examine the mRNA expression of O6-methylguanine DNA methyltransferase (MGMT) in normal and SBA spinal cords. MGMT promoter methylation was analysed using bisulfite sequencing PCR (BSP). Statistical analyses were performed using Student’s t-test and one-way Analysis of Variance (ANOVA). Our data showed that in normal controls, the MGMT mRNA expression was decreased with increasing embryonic days, and decreased dramatically on E11 to E14 (P=0.0483) and reached a minimum on E18 (vs E11, P=0.0031). In SBAs, γ-H2A.X was significantly increased (P=0.0174), and the mRNA expression of MGMT was significantly descreased on E14, E16 and E18 (P=0.0029, 0.0425 and 0.0386, respectively). The BSP results showed that almost all CpG sites in the MGMT promoter remained unmethylated in both SBAs and controls and no significant difference was detected between the two groups in either E14 or E18 embryos. Our results showed that DNA damage occurred in ATRA-induced SBA rat foetuses. The mRNA expression of MGMT is down-regulated in ATRA-induced SBA foetuses, and this down-regulation is independent of promoter DNA methylation.

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    Validity and Reliability of the Ocular Motor Nerve Palsy Scale
    Ling-Yun Zhou, Chang Su, Tie-Juan Liu, Xue-Mei Li
    2018, 13 (0):  1. 
    Abstract ( 115 )   PDF (820KB) ( 341 )   Save

    Objective and accurate assessment of the degree of ocular motor nerve palsy is helpful not only in the evaluation of prognosis, but also for the screening of treatment methods. However, there is currently no comprehensive measure of its severity. In this study, we designed the Ocular Motor Nerve Palsy Scale and investigated its validity and reliability. Six experts were invited to grade and evaluate the scale.The study recruited 106 patients with a definite diagnosis of unilateral isolated ocular motor nerve palsy. Three physicians evaluated the patients using the scale. One of the three physicians evaluated the patients again after 24 hours. The content validity index (CVI) and factor analysis were used to analyze the scale’s construct validity. The intraclass correlation coefficient and Cronbach’s alpha were used to evaluate the inter-rater and test-retest reliability and the internal consistency. The CVI results (I-CVI = 1.0, S-CVI = 0.9, Pc = 0.016, K* =1) indicated good content validity. Factor analysis extracted two common factors that accounted for 85.2% of the variance. Furthermore,the load value of each component was above 0.8, indicating good construct validity. The Ocular Motor Nerve Palsy Scale was found to be highly reliable, with an inter-rater reliability intraclass correlation coefficient of 0.965 (P < 0.01), a test-retest reliability intraclass correlation coefficient of 0.976 (P < 0.01), and Cronbach’s alpha values of 0.63–0.70. In conclusion, the Ocular Motor Nerve Palsy Scale with good validity and reliability can be used to quantify the severity of ocular motor nerve palsy.
    This study was registered at Chinese Clinical Trial Registry (registration number: ChiCTR-OOC-17010702).

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