Neural Regeneration Research ›› 2024, Vol. 19 ›› Issue (9): 1861-1862.doi: 10.4103/1673-5374.390977

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Does MgSO4 protect the preterm brain? Dissecting its role in the pathophysiology of hypoxic ischemic encephalopathy

Robert Galinsky*, Laura Bennet, Alistair J. Gunn#br#   

  1. The Ritchie Centre, Hudson Institute of Medical Research, Melbourne, VIC, Australia (Galinsky  R)
    Department of Obstetrics and Gynaecology, Monash University, Melbourne, VIC, Australia (Galinsky R)
    Department of Physiology, The University of Auckland, Auckland, New Zealand (Bennet L, Gunn AJ)
  • Online:2024-09-15 Published:2024-01-25
  • Contact: Robert Galinsky, PhD, Robert.Galinsky@hudson.org.au.
  • Supported by:
    This work was supported by Health Research Council of New Zealand (grants 17/601 and 22/559), the Auckland Medical Research Foundation, the Lottery Health Grants Board of New Zealand, the C. J. Martin Postdoctoral Fellowship and project grant from the National Health and Medical Research Council of Australia (APP1090890 and APP1164954) and the Victorian Government’s Operational Infrastructure Support Program (to RG).

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Abstract: Mitigating preterm encephalopathy continues to be one of the greatest challenges in perinatal medicine. Preterm encephalopathy is associated with high mortality, serious morbidity, and significant socio-economic impacts on the individuals, their families, and public health sectors and welfare systems that last a lifetime. The cost of disability associated with preterm brain injury continues to rise. Prevention of this injury, and disability, would significantly reduce this socioeconomic burden.